A novel drug delivery system of oral curcumin markedly improves efficacy of treatment for heart failure after myocardial infarction in rats.

نویسندگان

  • Yoichi Sunagawa
  • Hiromichi Wada
  • Hidetoshi Suzuki
  • Hiroki Sasaki
  • Atsushi Imaizumi
  • Hiroyuki Fukuda
  • Tadashi Hashimoto
  • Yasufumi Katanasaka
  • Akira Shimatsu
  • Takeshi Kimura
  • Hideaki Kakeya
  • Masatoshi Fujita
  • Koji Hasegawa
  • Tatsuya Morimoto
چکیده

Curcumin is an inhibitor of p300 histone acetyltransferase activity, which is associated with the deterioration of heart failure. We reported that native curcumin, at a dosage of 50 mg/kg, prevented deterioration of the systolic function in rat models of heart failure. To achieve more efficient oral pharmacological therapy against heart failure by curcumin, we have developed a novel drug delivery system (DDS) which markedly increases plasma curcumin levels. At the dosage of 0.5 mg/kg, DDS curcumin but not native curcumin restored left ventricular fractional shortening in post-myocardial infarction rats. Thus, our DDS strategy will be applicable to the clinical setting in humans.

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عنوان ژورنال:
  • Biological & pharmaceutical bulletin

دوره 35 2  شماره 

صفحات  -

تاریخ انتشار 2012